Research Radartracking 4 published studies · 1 human · 2 clinical trials · 2 cancer pages · updated Jun 2026Open the Research Map →

Vitamin C (IV/high dose)

IV ascorbate: H₂O₂/DNA ↑, TET ↑, sensitizer; early signals in pancreatic/ovarian/colorectal/lung.

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👥⭐⭐⭐ Early Clinical — Promising phase I/II signals and case series; larger randomized trials ongoing/needed.Ascorbate IVPharmacologic vitamin C

Forms: IV infusion (15-75 g in saline, 1-3x/week) · Oral high-dose (3-10 g/day, non-pharmacologic)

Educational only, not medical advice. OncoForge makes no claim that Vitamin C (IV/high dose) treats, prevents, or cures any condition, beyond what the linked studies show. Evidence levels vary; effects may not translate to people, and some compounds can cause harm. Always coordinate with your oncology team.

Key Takeaway

Pharmacologic ascorbate (≥15–50 g IV) reaches millimolar plasma levels that generate extracellular H₂O₂, creating tumor-biased oxidative stress and DNA damage, while also acting as a TET cofactor to favor demethylation. Early human trials show feasibility, symptom relief, and signals of synergy with chemotherapy/radiation in select cancers.

Evidence at a glance

Tier 3 · early humanPancreaticOvarianColorectalLung

Phase I/II pancreatic (Welsh) PFS signals; ovarian case series; meta-QoL modest; PD H₂O₂/γH2AX robust; ongoing NCT03146962 (combo); G6PD mandatory.

How it may work

Tumor-selective H₂O₂ formation exploits relatively low catalase/peroxidase buffering in cancer cells → oxidative DNA lesions, ATP depletion, and apoptosis/necrosis. Ascorbate supports α-KG–dependent dioxygenases (e.g., TETs), promoting demethylation and differentiation programs. Additional data suggest immune modulation and anti-angiogenic effects, plus chemo-/radiosensitization via redox cycling.

Targets & pathways

Curated mechanistic targets reported for this agent — how it may act on cells, not proof of a clinical effect.

  • H₂O₂Tumor-selective generation
  • DNA DamageOxidative lesions and DSBs
  • TET DemethylationCofactor for epigenetic enzymes
  • Chemo/RT SensitizerRedox cycling enhancement
  • ApoptosisCaspase activation
H₂O₂DNA DamageTET DemethylationChemo/RT Sensitizer

Often studied / combined with

Combinations reported in the literature, not a protocol or a recommendation.

Overlapping mechanisms

Safety & interactions

Severity and how well-established each signal is are shown separately. Verify everything with your oncologist or pharmacist — absence here does not mean safe.

Risk categories
G6pd DeficiencyVein IrritationGlucose Confound
Potential interactions
  • redox_chemoMonitorModerateTheoreticalSequence to avoid antagonism (e.g., bortezomib).
  • radiationSynergizeLowTheoreticalEnhanced ROS without added toxicity.
  • GemcitabineSynergizeLowTheoreticalImproved PFS in pancreatic trials.

Timing

References

Research

No published studies for Vitamin C (IV/high dose) yet

New studies appear here once they’ve been reviewed. Browse all studies.

Dose: as studied, not a recommendation

These are doses as studied or reported, never a recommendation. The right amount of Vitamin C (IV/high dose) depends on you, your other medicines, and your situation; decide it with your oncology team and pharmacist, not from a web page.

Ranges seen in adjunct / practice use: 15–75 g IV 1-3x/week (IV) Over 1-2h infusion; G6PD screen first, Trials 50-75 g 2-3x/week; adjust for body weight; oral 3-10 g/day supplemental..

Trials studying Vitamin C (IV/high dose)

Loading current trials from ClinicalTrials.gov… Search ClinicalTrials.gov →

Appears in these protocol claims

Vitamin C (IV/high dose) is named in these protocols discussed online. Listed for transparency: being part of a protocol is not evidence that it works, and OncoForge does not endorse them.

Inclusion here is not an endorsement. OncoForge makes no claim beyond what the linked studies show. Discuss anything on this page with your oncology team before acting on it.

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