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Leiomyosarcoma

A plain-English summary of the published research on Leiomyosarcoma, reviewed and approved by our editors — not a hand-curated clinical overview.

Research summary · reviewed
Educational only: This page is not medical advice. Coordinate decisions with your oncology team.

Reviewed Jun 2026 · OncoForge editorial · How we review →

AI extractedhuman reviewedsources checkedretractions suppressed· last updated Jun 2026

Evidence at a glanceHuman · observationalMixed results⚠ Studies disagree
62 published studies that name Leiomyosarcoma14 human studies approved & graded (trial, observational, or meta-analysis)72 human clinical studies in the Leiomyosarcoma corpus817 source documents in the Leiomyosarcoma corpus

last checked June 19, 2026

Why this grade?

Human · observationalHuman observational evidence only — no trials.

Computed deterministically from the studies’ types and reported outcomes — not written by AI, and not a claim that anything works.

What the guidelines say

NCI PDQESMONCCNASCO

We link the authoritative guidelines rather than reproduce them. Below, the treatments on this page are split into standard care, guideline or regulatory options, supportive care, and studied but not standard so established care is not mixed with experimental or supportive items.

Studied, not standard - investigational
  • adjuvant chemotherapy (various regimens)
  • adjuvant radiotherapy
  • gemcitabine/docetaxel regimen
  • lymphadenectomy
  • radical or partial cystectomy
  • Cisplatin
  • Olaparib
  • Trastuzumab Deruxtecan
  • Trastuzumab-Deruxtecan (T-Dxd)
  • Bevacizumab
  • Dacarbazine
  • Doxorubicin
  • Gemcitabine
  • Ifosfamide
  • Nivolumab †Rx
  • Pembrolizumab

Read the guidelines

Cancer-specific deep links aren’t curated yet — these search the authoritative sources for Leiomyosarcoma.

Treatment map: Leiomyosarcoma

Open as a full page →

Standard care plus every compound studied in the literature (each cited) and graded by evidence, organized by clinical readiness. A category, not a verdict that anything works — confirm anything here with your oncology team.

16
Interventions
0
Standard of care
9
Tested in people
7
Lab / animal
0
Named in lit.
6
Classes
Standard of care (0) Guideline option (0) Tested in people (9) Lab / animal only (7) Named in the literature (0)

Tested in people, by trial phase: phase not reported ×9

Clinical evidence
Preclinical evidence
Standard of care
Guideline option
Tested in people
Lab / animal only
Named in the literature
Surgery & procedures
2
Radiotherapy
1
Chemotherapy
3
2
Targeted therapy
3
1
Immunotherapy
2
Other
2

Columns group into clinical evidence (used in, or tested on, people) and preclinical evidence (lab/animal, or only named in the literature). Cell = number of interventions; a dashed cell means none recorded there.

Investigational & adjunct compounds — detail (16)
Meta-analysis (5)
adjuvant chemotherapy (various regimens)· Adjuvant (after surgery)adjuvant radiotherapy· Adjuvant (after surgery)gemcitabine/docetaxel regimen· Adjuvant (after surgery)lymphadenectomyradical or partial cystectomy

"Tested in people" rows show the highest trial phase found in that compound's cited human studies (Phase I–IV; "phase not reported" = a human study with no phase tag). "Studied" = named in the cited literature for this cancer. "FDA ✓" = FDA-approved for this cancer; "off-label" = an FDA-approved drug used outside its approved indications (per openFDA). Not a claim that anything works.

Reported figures

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Snapshot

The essentials in ~60 seconds — every line is drawn from the cited sources below.

What it is
A type of uterine sarcoma listed among WHO uterine tumor categories; it is rare and genetically diverse and is staged using the 2014 FIGO and 2010 AJCC TNM systems. [1]
Survival
In a pooled series of urinary bladder leiomyosarcoma, 5‑year and 10‑year cancer-specific cumulative mortality were 38% and 50%, respectively. [2]
Standard treatment
Surgery is the primary approach; a meta-analysis found that adjuvant chemotherapy and adjuvant radiotherapy did not decrease recurrence risk in surgically treated early-stage uterine leiomyosarcoma, and systematic lymphadenectomy showed little prognostic or therapeutic benefit except for obvious extrauterine involvement or suspicious nodes. [3][4]
Biggest challenge
Because uterine leiomyosarcoma is rare and genetically diverse, there is a lack of consensus on risk factors and optimal therapeutic choices, and reported molecular subgroups are based on limited case numbers requiring further study. [1][5]

Ask about Leiomyosarcoma

Answers come only from the cited sources on this page — with the supporting evidence shown. If the sources here don't cover your question, it will say so. Educational information, not medical advice.

Key numbers & factors

Biomarkers

  • Hormone receptors, cytokeratin, smooth muscle markers (polyphenotypic immunophenotype) · Characteristic co‑expression pattern of UTROSCT [5]
  • FOXL2 protein · Reported positive in UTROSCT but FOXL2 and DICER1 mutations were not identified in reported studies [5]
  • ESR1-NCOA2/3, GREB1-NCOA1/2, GREB1-CTNNB1 gene fusions · Reported gene fusions in UTROSCT suggesting distinct molecular subgroups [5]
  • SF-1 · May help differentiate UTROSCT from other uterine lesions with sex cord–like architecture, but reported expression rates vary between series [5]

9 sections — tap any heading to expand its cited detail. Key points are above.

Overview1 point
  • Leiomyosarcoma is listed among the World Health Organization categories of uterine tumor types. [1]
EpidemiologyUterine sarcomas represent a small fraction of uterine cancers. In a pooled series of adult urinary bladder leiomyosarcoma cases the mean age was 52 years and most tumors were high-grade.3 points
  • Uterine sarcomas account for 3-7% of all uterine cancers. [1]
  • In a pooled series of adult urinary bladder leiomyosarcoma cases the mean age of patients was 52 years. [2]
  • In that urinary bladder leiomyosarcoma series the majority (75%) of tumors were classified as high-grade sarcomas. [2]
Key biomarkersUterine tumor resembling ovarian sex cord tumor (UTROSCT) shows a polyphenotypic immunophenotype with co-expression of hormone receptors, cytokeratin, smooth muscle markers and markers seen in ovarian sex cord–stromal tumors, and has been reported to harbor recurrent gene fusions such as ESR1-NCOA2/3, GREB1-NCOA1/2 and GREB1-CTNNB1.4 points
  • UTROSCT characteristically exhibits a polyphenotypic immunophenotype with co‑expression of hormone receptors, cytokeratin, smooth muscle markers, and several markers commonly positive in ovarian sex cord–stromal tumors. [5]
  • Although FOXL2 protein positivity has been reported in UTROSCT, FOXL2 and DICER1 mutations were not identified in this tumor in reported studies. [5]
  • UTROSCT has been reported to contain gene fusions including ESR1-NCOA2/3, GREB1-NCOA1/2 and GREB1-CTNNB1, as well as other chromosomal translocations. [5]
  • One study reported SF-1 showed a specificity of 100% for differentiating UTROSCT from other uterine lesions with sex cord-like architectures; however, SF-1 expression was reported as 57.9% and 50.0% in two small series, indicating low expression rates in some reports. [5]
Biology & pathways1 point
  • GREB1 and ESR1 gene rearrangements and specific chromosomal translocations have been described in UTROSCT, suggesting distinct molecular subgroups. [5]
Standard managementMeta-analyses indicate that adjuvant chemotherapy and adjuvant radiotherapy did not decrease the risk of recurrence after surgery for early-stage uterine leiomyosarcoma. A separate meta-analysis of uterine sarcoma studies found that lymphadenectomy provided little prognostic or therapeutic benefit and may not be recommended unless there is obvious extrauterine involvement or suspicious nodes.2 points
  • Meta-analysis evidence suggests that adjuvant chemotherapy and adjuvant radiotherapy did not decrease the risk of recurrence in surgically treated early-stage uterine leiomyosarcoma. [3]
  • The meta-analysis suggests lymphadenectomy bears little prognostic or therapeutic benefit in uterine sarcoma, and systematic lymphadenectomy may not be recommended unless there is obvious extrauterine involvement, clinically suspicious enlarged nodes, or advanced sarcomas. [4]
Treatments & compounds studiedFive therapeutic approaches are reported across chemotherapy, radiotherapy, and procedural/surgical interventions.5 treatments

Chemotherapy

  • adjuvant chemotherapy (various regimens): Adjuvant (after surgery)A meta-analysis in early-stage uterine leiomyosarcoma found that adjuvant chemotherapy did not decrease the risk of recurrence compared with observation. [3]
  • gemcitabine/docetaxel regimen: Adjuvant (after surgery)A pooled analysis examined a gemcitabine/docetaxel regimen within subgroup analyses and reported that neither adjuvant chemotherapy nor adjuvant radiotherapy decreased recurrence significantly in those subgroup analyses. [3]

Radiotherapy

  • adjuvant radiotherapy: Adjuvant (after surgery)A meta-analysis in early-stage uterine leiomyosarcoma found that adjuvant radiotherapy did not decrease the risk of recurrence compared with observation. [3]

Procedures & devices

  • lymphadenectomy: A pooled analysis reported a pooled relative risk for overall survival of 0.90 (95% CI, 0.62-1.31) for uterine leiomyosarcoma patients who underwent lymphadenectomy, with no significant association with improved overall survival. [4]
    RR 0.9 (95% CI 0.62–1.31) vs no lymphadenectomy
    Source quote
    • The pooled RR for uterine leiomyosarcoma (uLMS) in patients with LAD in 5 trials was 0.90 (95% CI, 0.62-1.31) and for endometrial stromal sarcoma (ESS) in 11 trials was 0.96 (95% CI, 0.69-1.34), suggesting that there was no significant benefit of LAD in improving overall survival (P < 0.05).
  • radical or partial cystectomy: A systematic review of urinary bladder leiomyosarcoma concluded that surgery (radical or partial cystectomy) with negative resection margins appears to be the most promising option, possibly supplemented by chemotherapy or radiation. [2]
Staging & risk1 point
  • Staging of uterine leiomyosarcoma is done using the 2014 FIGO system and the 2010 AJCC TNM system according to a systematic review. [1]
PrognosisIn a pooled series of urinary bladder leiomyosarcoma, 5‑year and 10‑year cancer-specific cumulative mortality rates were reported as 38% and 50%, respectively. In that series, patients with high-grade sarcomas had a trend toward higher mortality compared with low-grade tumors (p = 0.0280).2 points

Key figures

Survival & outcomes
OutcomeValue95% CI
5-year cancer-specific cumulative mortality rate38%
10-year cancer-specific cumulative mortality rate50%
Source quotes
  • For the whole sample, we determined 5- and 10-year cancer-specific cumulative mortality rates of 38 and 50%.
  • In the pooled series of urinary bladder leiomyosarcoma, 5‑year and 10‑year cancer-specific cumulative mortality rates were reported as 38% and 50%, respectively. [2]
  • In that bladder leiomyosarcoma series patients with high-grade sarcomas had a trend toward higher mortality compared with low-grade tumors (p = 0.0280). [2]
What we don't know yetUterine leiomyosarcoma is rare and genetically diverse, contributing to limited consensus about risk factors and optimal therapeutic choices. Data on specific molecular subtypes are sparse—for example, reports of GREB1‑rearranged uterine tumors note small case numbers and call for further investigation.2 points
  • Because uterine leiomyosarcoma is rare and genetically diverse, there is a lack of consensus on risk factors and optimal therapeutic choices. [1]
  • Authors reporting GREB1-rearranged uterine tumors stated that the number of cases was limited and that further investigation is required. [5]

Common questions

How common is Leiomyosarcoma?

Uterine sarcomas represent a small fraction of uterine cancers. In a pooled series of adult urinary bladder leiomyosarcoma cases the mean age was 52 years and most tumors were high-grade.

Which biomarkers are important in Leiomyosarcoma?

Uterine tumor resembling ovarian sex cord tumor (UTROSCT) shows a polyphenotypic immunophenotype with co-expression of hormone receptors, cytokeratin, smooth muscle markers and markers seen in ovarian sex cord–stromal tumors, and has been reported to harbor recurrent gene fusions such as ESR1-NCOA2/3, GREB1-NCOA1/2 and GREB1-CTNNB1.

How is Leiomyosarcoma treated?

Meta-analyses indicate that adjuvant chemotherapy and adjuvant radiotherapy did not decrease the risk of recurrence after surgery for early-stage uterine leiomyosarcoma. A separate meta-analysis of uterine sarcoma studies found that lymphadenectomy provided little prognostic or therapeutic benefit and may not be recommended unless there is obvious extrauterine involvement or suspicious nodes.

What treatments are studied for Leiomyosarcoma?

Five therapeutic approaches are reported across chemotherapy, radiotherapy, and procedural/surgical interventions.

What is the prognosis for Leiomyosarcoma?

In a pooled series of urinary bladder leiomyosarcoma, 5‑year and 10‑year cancer-specific cumulative mortality rates were reported as 38% and 50%, respectively. In that series, patients with high-grade sarcomas had a trend toward higher mortality compared with low-grade tumors (p = 0.0280).

What is still being researched in Leiomyosarcoma?

Uterine leiomyosarcoma is rare and genetically diverse, contributing to limited consensus about risk factors and optimal therapeutic choices. Data on specific molecular subtypes are sparse—for example, reports of GREB1‑rearranged uterine tumors note small case numbers and call for further investigation.

Sources

Every statement above is drawn from these reviewed sources. This page reports what they describe. Sources last checked June 19, 2026.

  1. Systematic reviewUterine sarcoma Part I-Uterine leiomyosarcoma: The Topic Advisory Group systematic review · 2016
  2. Meta-analysisLeiomyosarcoma of the Urinary Bladder in Adult Patients: A Systematic Review of the Literature and Meta-Analysis · 2019
  3. Meta-analysisEffect of adjuvant therapy on the risk of recurrence in early-stage leiomyosarcoma: A meta-analysis · 2019
  4. Meta-analysisRole of Lymphadenectomy for Uterine Sarcoma: A Meta-Analysis · 2017
  5. Systematic reviewUterine lesions with sex cord-like architectures: a systematic review · 2019

What supports this page

The kinds of sources behind this page, strongest at the top. Faint rungs show what is not here yet.

Guideline
4
Meta-analysis
20
Systematic review
38
Randomized trial
1
Clinical trial
15
Observational
2
Case report
295
Review
426
Preclinical
0
Other
16

Living document — last change June 19, 2026: Cancer page updated. 2 recent updates logged.

Pooled evidence across studies

PubMed
  • Largest specimen weights: 3183 g (2933–4780 across studies) · (regimen unspecified)
    3 studies · 67% agree · moderate · 1 flagged37297823
  • 5-year RFS: 85% (69–93 across studies) · (regimen unspecified)
    3 studies · 67% agree · moderate32537765
  • OS: 18.7 months (6–28 across studies) · platinum-based chemotherapy + surgery + chemotherapy + radiation
    3 studies · 33% agree · heterogeneous21740740
  • 1-year OS: 58% (29–71 across studies) · platinum-based chemotherapy + surgery + chemotherapy + radiation
    3 studies · 33% agree · heterogeneous21740740
  • Pattern of recurrence in early-stage disease: 13.5 (9–18 across studies) · (regimen unspecified)
    2 studies · 0% agree · heterogeneous40044476

Compounds compared by evidence

PubMed

How to read this: Ranked by the strength and volume of the evidence — NOT by how well a treatment works. A higher rank means a compound has been studied more, or in stronger study designs (e.g. randomized trials over lab studies), not that it produces better outcomes. The effect column shows the largest pooled figure reported, not a head-to-head comparison.

#CompoundEvidence strengthStudiesLargest pooled effect
1Cisplatin ChemotherapyHuman · observational1
2Olaparib Targeted therapyHuman · observational1
3Trastuzumab Deruxtecan Targeted therapyHuman · observational1
4Trastuzumab-Deruxtecan (T-Dxd) Targeted therapyHuman · observational1
5Dacarbazine OtherInsufficient evidence1
6Doxorubicin ChemotherapyInsufficient evidence1
7Gemcitabine ChemotherapyInsufficient evidence1
8Ifosfamide OtherInsufficient evidence1

Medicines & supplements studied for Leiomyosarcoma

PubMedFDAClinicalTrials.gov

Every drug, supplement, and other agent the published studies cover for Leiomyosarcoma, ranked by how strong the evidence is — what studies report, not a recommendation. Tap any to see its full profile.

Medicines · 11

CisplatinHuman · observationalReported positive1 human

Human observational evidence only — no trials.

Largest credible effect: Individuals with HRD uLMS in the screened cohort 9%, n=58 PMID 37143137 · effect sizes 2–13 across 4 studies

Most authoritative study: Targeting homologous recombination deficiency in uterine leiomyosarcoma

Based on a single study.
ChemotherapyFDA off-label1 studyFull profile →
OlaparibHuman · observationalReported positive1 human

Human observational evidence only — no trials.

Largest credible effect: Individuals with HRD uLMS in the screened cohort 9%, n=58 PMID 37143137 · effect sizes 2–13 across 4 studies

Most authoritative study: Targeting homologous recombination deficiency in uterine leiomyosarcoma

Based on a single study.
Targeted therapyFDA off-label1 studyFull profile →
Trastuzumab DeruxtecanHuman · observationalReported positive1 human

Human observational evidence only — no trials.

Largest credible effect: median PFS 5.4 mo [0.8–9.8], n=10 PMID 39639215 · effect sizes 1–5 across 3 studies

Most authoritative study: Real-world evidence of Trastuzumab Deruxtecan (T-DXd) Efficacy in HER2-expressing gynecological malignancies

Based on a single study.
Targeted therapyFDA off-label1 studyFull profile →
Trastuzumab-Deruxtecan (T-Dxd)Human · observationalReported positive1 human

Human observational evidence only — no trials.

Largest credible effect: median PFS 5.4 mo [0.8–9.8], n=10 PMID 39639215 · effect sizes 1–5 across 3 studies

Most authoritative study: Real-world evidence of Trastuzumab Deruxtecan (T-DXd) Efficacy in HER2-expressing gynecological malignancies

Based on a single study.
Targeted therapy1 studyFull profile →
BevacizumabInsufficient evidenceInconclusive

No primary experimental studies yet.

Largest credible effect: objective response (complete + partial) 35%, n=69 PMID 29759566 · response rates 11–35 across 4 studies

Most authoritative study: Role of bevacizumab in uterine leiomyosarcoma

No human studies yet · Based on a single study.
Targeted therapyFDA off-label1 studyFull profile →
DacarbazineInsufficient evidenceMixed results

No primary experimental studies yet.

Most authoritative study: Management of advanced uterine leiomyosarcoma

No human studies yet · No numeric effect sizes reported · Based on a single study.
OtherFDA off-label1 studyFull profile →
DoxorubicinInsufficient evidenceMixed results

No primary experimental studies yet.

Most authoritative study: Management of advanced uterine leiomyosarcoma

No human studies yet · No numeric effect sizes reported · Based on a single study.
ChemotherapyFDA off-label1 studyFull profile →
GemcitabineInsufficient evidenceReported negative

No primary experimental studies yet.

Largest credible effect: chemotherapy_duration 6 mo, n=1 PMID 39286855 · effect sizes 6–8 across 2 studies

Most authoritative study: Primary ovarian leiomyosarcoma: a case report

No human studies yet · Based on a single study.
ChemotherapyFDA off-label1 studyFull profile →
IfosfamideInsufficient evidenceMixed results

No primary experimental studies yet.

Most authoritative study: Management of advanced uterine leiomyosarcoma

No human studies yet · No numeric effect sizes reported · Based on a single study.
OtherFDA off-label1 studyFull profile →
Nivolumab †RxInsufficient evidenceMixed results

No primary experimental studies yet.

Most authoritative study: Uterine leiomyosarcoma: A review of the literature and update on management options

No human studies yet · No numeric effect sizes reported · Based on a single study.
Immunotherapy1 studyFull profile →
PembrolizumabInsufficient evidenceMixed results

No primary experimental studies yet.

Most authoritative study: Uterine leiomyosarcoma: A review of the literature and update on management options

No human studies yet · No numeric effect sizes reported · Based on a single study.
ImmunotherapyFDA off-label1 studyFull profile →

What recent studies report in Leiomyosarcoma

These are reviewed studies whose abstracts concern Leiomyosarcoma. Each describes only what that study reported. This is not a claim by OncoForge that any compound helps or harms Leiomyosarcoma. Most are early lab, animal, or small human studies, and findings often conflict.

60 studies12 human⚠ Conflicting evidenceMechanism (17)Trial (2)Safety (1)

Tracking 60 published studies of Leiomyosarcoma: 12 in humans, 48 reviews/other.

Reported direction across studies: 11 positive, 19 mixed, 6 negative, 24 inconclusive.

Findings conflict — both supportive and negative/mixed results exist (see below). Human evidence is limited.

These counts summarize what the studies reported; they are not a measure of whether anything works for Leiomyosarcoma.

Compounds with studies mentioning Leiomyosarcoma

Trastuzumab deruxtecan t dxd (1)Trastuzumab deruxtecan (1)Gemcitabine (1)Olaparib (1)Cisplatin (1)Nivolumab (1)Pembrolizumab (1)Bevacizumab (1)Doxorubicin (1)Ifosfamide (1)Dacarbazine (1)
ReviewMechanismMixed resultsLimited evidenceTier 4 · clinical

Immune landscape and potential role of immune checkpoint inhibitors on uterine leiomyosarcoma: a review

International journal of gynecological cancer : official journal of the International Gynecological Cancer Society · Jan 2026

uterine leiomyosarcoma

This is a narrative review of the immune environment of uterine leiomyosarcoma and the potential role of immune checkpoint inhibitors. The authors report variable PD-L1 expression, heterogeneous lymphocytic infiltration, and interactions with tumor-associated macrophages, note modest response rates to checkpoint inhibitors in clinical trials, and discuss that dual PD-1/CTLA-4 blockade and chemotherapy-induced immunogenic cell death may enhance immune activation in select patients. They conclude that combinatorial and personalized strategies, improved immune profiling, and macrophage-targeted approaches merit further study.

Studied with: anti-CTLA-4 + anti-PD-1 dual checkpoint blockade, chemotherapy (to induce immunogenic cell death), targeted immunomodulation, macrophage-targeted therapies.

Key findings
  • Uterine leiomyosarcoma has a variable tumor immune microenvironment including variable PD-L1 expression and differential lymphocytic infiltration.
  • Interactions with tumor-associated macrophages shape immune responses in leiomyosarcoma.
  • Clinical trials of immune checkpoint inhibitors in leiomyosarcoma have produced modest response rates.
  • Molecular analyses suggest that specific sub-groups of leiomyosarcoma patients may derive greater benefit from checkpoint inhibition.
  • Dual-checkpoint blockade combining anti-PD-1 and anti-CTLA-4 has demonstrated enhanced immune activation in select patients.
  • Chemotherapy-induced immunogenic cell death has been explored as a complementary approach to immunotherapy.
  • Authors recommend innovative combinatorial strategies, improved patient selection, enhanced macrophage-targeted therapies, and optimized immune profiling for future research.
Limitations: Narrative review only—no new primary experimental or patient-level data are presented.; Conclusions are based on heterogeneous published studies and molecular analyses rather than definitive randomized trial evidence.; Abstract reports only 'modest response rates' in trials without quantitative pooled estimates or meta-analysis.; Leiomyosarcoma heterogeneity and rarity limit generalizability of existing trial results (as noted by authors)..

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed

Human · observationalMixed resultsModerate evidenceTier 3 · early humann = 661

Prognosis and Follow-Up Recommendations for Subcutaneous and Dermal Leiomyosarcoma: Local Recurrence, Metastasis, and Overall Survival in a Danish Nationwide Cohort of 661 Patients

Journal of surgical oncology · Nov 2025 · nationwide population-based cohort study (patients diagnosed 1980–2022)

cutaneous leiomyosarcomasubcutaneous leiomyosarcomadermal leiomyosarcomaleiomyosarcomaskin neoplasms

This nationwide Danish cohort study (1980–2022) compared outcomes in 196 patients with subcutaneous and 465 patients with dermal cutaneous leiomyosarcoma (total n=661). At 10 years the local recurrence rates were similar (15% vs 11%, p=0.13), but subcutaneous tumors had a much higher 10-year metastasis risk (25% vs 2.7%, p<0.001) and lower 10-year overall survival (56% vs 64%, p=0.02). The authors conclude that grade 2–3 subcutaneous leiomyosarcoma should be considered high-risk and recommend 5 years of follow-up including clinical exam plus PET/CT or chest CT, while dermal leiomyosarcoma can have clinical follow-up for 4 years.

Reported effects: 10-year local recurrence 15%, p 0.13, n=196 · 10-year risk of metastasis 25%, p <0.001, n=196 · +1 more

Key findings
  • Cohort included 196 patients with subcutaneous leiomyosarcoma and 465 with dermal leiomyosarcoma (total n=661).
  • The 10-year local recurrence rate was similar: subcutaneous 15% and dermal 11% (p = 0.13).
  • The 10-year risk of metastasis was substantially higher for subcutaneous leiomyosarcoma (25%) versus dermal (2.7%), p < 0.001; metastases were primarily observed in grade 2 and 3 tumors.
  • Ten-year overall survival was lower for subcutaneous compared with dermal leiomyosarcoma (56% vs. 64%), p = 0.02.
  • Authors recommend classifying grade 2–3 subcutaneous leiomyosarcoma as high-risk and performing clinical examinations plus PET/CT or CT thorax for 5 years; dermal leiomyosarcoma follow-up can focus on clinical exams for 4 years given very low metastasis risk.
Limitations: Observational, registry-based cohort (potential for residual confounding and unmeasured variables).; Long study period (1980–2022) during which diagnostic methods, staging, and treatments likely changed, which may affect outcomes.; Abstract does not report details on treatments, adjuvant therapy, or timing of interventions that could influence recurrence, metastasis, or survival.; Potentially incomplete or variable clinical data quality across decades and centers (not detailed in abstract).; Findings are from Denmark and may not generalize to other healthcare settings or populations..

Provides 10-year estimates of local recurrence, metastasis, and overall survival for dermal versus subcutaneous cutaneous leiomyosarcoma and gives follow-up recommendations.

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed · Full text

Human · observationalMechanismMixed resultsLimited evidenceTier 3 · early humann = 39

Primary ovarian leiomyosarcoma: results from an analysis by the French Sarcoma Group (Ovarian SArcoma MAnagement - OSAMA Study)

International journal of gynecological cancer : official journal of the International Gynecological Cancer Society · Apr 2025 · retrospective multicenter study using the national NetSarc database

primary ovarian leiomyosarcomaovarian neoplasmsleiomyosarcoma

This retrospective multicenter study analyzed 39 patients with primary ovarian leiomyosarcoma from the French Sarcoma Group database to describe clinical, surgical, pathological features and outcomes. Median tumor size was large (134 mm); 15 patients (44%) died of disease. Certain pathologic features (high mitotic counts, progesterone receptor negativity) were associated with worse survival. The authors conclude surgery is the mainstay for early-stage disease and the role of adjuvant therapy remains unclear.

Reported effects: total patients included 39 · localized disease - n 35 · +14 more

Key findings
  • 39 patients were included (35 localized, 4 metastatic).
  • Median tumor size was 134 mm.
  • Radical surgery was performed in 21 patients (62%) and wide surgery in 13 patients (38%).
  • Tumor grade 3 reported in 17 of 34 patients (50%); necrosis in 29 of 34 (85%); mitoses ≥20/HPF in 17 of 34 (50%); Ki-67 >30% in 17 of 27 patients (63%).
  • Estrogen receptor positive in 14 of 27 patients (52%); progesterone receptor positive in 10 of 27 patients (37%).
  • Adjuvant chemotherapy given in 12 of 34 patients (35%); pelvic adjuvant radiotherapy in 8 of 34 (23%).
  • Among early-stage cases, 9 had isolated pelvic recurrence and 18 had parenchymal distant metastases.
  • Fifteen patients (44%) died of disease.
  • High mitotic counts and progesterone receptor negativity were associated with worse survival in early-stage disease.
Limitations: Retrospective study design.; Small sample size (n=39) for subgroup and prognostic analyses.; Heterogeneous and incompletely reported treatment approaches (adjuvant therapies given in subsets).; Some pathological and biomarker data were available only in subsets (denominators vary: data reported for 34 or 27 patients), indicating missing data.; No control group or prospective follow-up protocol reported..

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed

Human · observationalReported positiveLimited evidenceTier 3 · early humann = 10

Real-world evidence of Trastuzumab Deruxtecan (T-DXd) Efficacy in HER2-expressing gynecological malignancies

BMC cancer · Dec 2024 · retrospective cohort

Trastuzumab-deruxtecan-t-dxdTrastuzumab-deruxtecanendometrial neoplasmsovarian neoplasmscervical neoplasmsuterine carcinosarcomauterine leiomyosarcomauterine serous carcinomaovarian carcinosarcomahigh-grade serous ovarian carcinomamucinous ovarian carcinomasquamous cervical carcinoma

This retrospective single-center study identified 10 patients with HER2-expressing (IHC 2+/3+) recurrent or metastatic gynecological cancers who received trastuzumab deruxtecan (5.4 mg/kg IV every 3 weeks). The cohort had a median progression-free survival of 5.4 months (95% CI 0.8-9.8). Five patients had a partial response, one had stable disease at 12 weeks, and four had disease progression at initial assessment. Clinical benefit was observed mainly in tumors with HER2 IHC 3+.

Reported effects: median PFS 5.4 mo [0.8–9.8], n=10 · partial responses 5, n=10 · +2 more

Key findings
  • 10 patients with recurrent/metastatic HER2-expressing gynecological malignancies were treated with T-DXd.
  • Histologies included uterine neoplasms (n=5), cervical squamous carcinoma (n=1) and ovarian cancers (n=4).
  • Median age was 65.4 years (25th-75th percentile, 58.1-75.2 years).
  • HER2 by IHC: 5 patients were 3+ and 5 patients were 2+.
  • Median number of prior therapy lines was 4 (range 2-6); 2 uterine serous carcinoma patients were pretreated with trastuzumab and 4 patients had prior immunotherapy.
  • Dose: T-DXd 5.4 mg/kg IV every 3 weeks until progression/toxicity.
  • Median progression-free survival (PFS) in the cohort was 5.4 months (95% CI 0.8-9.8 months).
  • Responses: 5 patients had partial response (including 2 previously treated with trastuzumab), 1 patient had stable disease at 12 weeks, 4 patients had disease progression at initial assessment.
  • Most patients who derived clinical benefit had HER2 IHC 3+ expression.
Limitations: Retrospective, single-center design; Very small sample size (n=10); No control or comparator arm; Heterogeneous mix of gynecologic histologies; Heavily pre-treated population limits generalizability; Limited/absent reporting of safety or adverse event data in the abstract; Potential selection and reporting bias inherent to retrospective series.

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed · Full text

Case reportReported negativeLimited evidenceTier 3 · early humann = 1

Primary ovarian leiomyosarcoma: a case report

The Journal of international medical research · Sep 2024 · case report

Gemcitabineovarian leiomyosarcoma

This is a case report of a woman in her late 50s diagnosed with primary ovarian leiomyosarcoma who underwent surgery. One month after surgery she received gemcitabine plus docetaxel chemotherapy for six months. Recurrence in the pelvic cavity was detected eight months after surgery. The report aims to raise awareness of this rare disease.

Reported effects: chemotherapy_duration 6 mo, n=1 · time_to_recurrence 8 mo, n=1

Studied with: gemcitabine + docetaxel.

Key findings
  • A woman in her late 50s presented with a 6-month history of abdominal pain and imaging revealed a pelvic mass.
  • She underwent surgery and was diagnosed with primary ovarian leiomyosarcoma.
  • One month postoperatively she began gemcitabine and docetaxel chemotherapy and continued this treatment for 6 months.
  • Eight months postoperatively recurrence was detected in the pelvic cavity.
Limitations: Single-patient case report (n=1), limiting generalizability; No chemotherapy doses or detailed regimen parameters provided; Short follow-up reported (recurrence at 8 months) with no long-term outcome data; No control or comparator group.

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed · Full text

ReviewMechanismInconclusiveLimited evidenceTier 4 · clinical

Molecular genetics and research progress of uterine leiomyosarcoma

Yi chuan = Hereditas · Aug 2024 · review

uterine leiomyosarcoma

This article is a review of the molecular pathology of uterine leiomyosarcoma (uLMS). The authors summarize molecular genetic features, epigenetic variants, experimental models, and clinical research progress, and note that understanding of uLMS pathogenesis is inadequate and disease models are limited, which hinders development of effective therapies.

Key findings
  • uLMS is an aggressive malignant soft-tissue tumor arising from the myometrium that is difficult to distinguish from benign leiomyoma in early stages and has a poor prognosis.
  • Current studies of uLMS pathogenesis and disease biology are inadequate.
  • uLMS disease models are very limited, which hinders development of effective therapeutics.
  • The review systematically summarizes molecular genetic features, epigenetic alterations, experimental models, and clinical research progress, and discusses directions including tumor evolution, the tumor microenvironment, and therapy development.
Limitations: This publication is a review and does not present new primary experimental or clinical data.; Abstract does not describe review methods in detail (e.g., search strategy, inclusion criteria).; Field-level limitations noted by the authors include inadequate pathogenesis studies and limited disease models, which constrain conclusions about therapeutic development..

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed

Case reportInconclusiveLimited evidenceTier 3 · early humann = 1

Rare case of giant ischiorectal leiomyosarcoma

International journal of surgery case reports · Aug 2024 · case report

leiomyosarcomasoft tissue sarcomaischiorectal leiomyosarcoma

This is a single-patient case report of a 25-year-old woman who presented with a left buttock swelling. MRI showed a large ischiorectal mass measuring 9 by 9.5 by 18 cm; the tumor was completely excised and immunohistochemistry confirmed leiomyosarcoma. The authors discuss diagnostic and therapeutic challenges due to the tumor's rarity and size and recommend individualized, multidisciplinary management.

Reported effects: tumor_dimension_1 9, n=1 · tumor_dimension_2 9.5, n=1 · +1 more

Key findings
  • Single case of ischiorectal leiomyosarcoma in a 25-year-old female presenting with a left buttock swelling.
  • MRI measured the tumor as 9 by 9.5 by 18 cm.
  • Complete surgical excision of the tumor was performed.
  • Immunohistochemistry confirmed the diagnosis of leiomyosarcoma.
  • Authors highlight rarity, diagnostic/therapeutic challenges, and recommend a multidisciplinary, individualized approach.
Limitations: Single-patient case report limits generalizability.; No long-term follow-up or outcomes (recurrence, survival) reported.; No control or comparative data to inform management decisions.; No details on surgical margins, adjuvant therapy, or postoperative course in the abstract..

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed · Full text

Case reportReported positiveLimited evidenceTier 3 · early humann = 1

Primary Sternal Leiomyosarcoma

Annals of thoracic surgery short reports · Jul 2024 · case report

primary sternal leiomyosarcoma

A 70-year-old man had an incidentally discovered sternal lesion on PET-CT performed for prostate cancer staging. Biopsy showed spindle cells suggesting probable leiomyosarcoma and contrast CT found no other primary site, suggesting a primary sternal tumor. The patient underwent sternotomy and reconstruction with methyl methacrylate and had an uneventful recovery. The authors note primary sternal leiomyosarcoma is very rare (<0.7% of primary malignant bone tumors).

Key findings
  • Lesion was incidentally found on PET-CT during prostate cancer staging.
  • Interventional radiology biopsy showed spindle cells indicating probable leiomyosarcoma.
  • Contrast CT found no other primary site, supporting the interpretation of a primary sternal tumor.
  • Patient underwent sternotomy and reconstruction with methyl methacrylate and recovered uneventfully.
  • Primary sternal leiomyosarcoma is rare, comprising less than 0.7% of primary malignant bone tumors.
Limitations: Single case report (n=1) limits generalizability.; Diagnosis described as 'probable' leiomyosarcoma from biopsy; the abstract does not state whether resection pathology confirmed the diagnosis.; No long-term follow-up or oncologic outcomes reported beyond an 'uneventful recovery.'; No information on margins, adjuvant therapy, or postoperative oncologic surveillance provided..

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed · Full text

ReviewMechanismMixed resultsLimited evidenceTier 4 · clinical

Comprehensive Review of Uterine Leiomyosarcoma: Pathogenesis, Diagnosis, Prognosis, and Targeted Therapy

Cells · Jun 2024

uterine leiomyosarcoma

This is a narrative review summarizing current knowledge about uterine leiomyosarcoma (uLMS), including its poor prognosis and high rates of recurrence and metastasis. The authors discuss multiple biological pathways implicated in uLMS (DNA repair, immune checkpoints, protein kinases, hedgehog), the emerging roles of epigenetics and the epitranscriptome, biomarkers and diagnostic approaches (including AI and shear wave elastography), current medical management, and ongoing clinical trials. The abstract notes that drugs targeting abnormal pathway functions have been reported to improve survival but that chemotherapy resistance remains a major challenge.

Key findings
  • uLMS has a poor prognosis with high rates of recurrence and metastasis; five-year survival reported between 25 and 76%, and survival approaches 10-15% for patients with metastatic disease at initial diagnosis.
  • Multiple biological pathways are implicated in uLMS pathogenesis, including DNA repair defects, immune checkpoint pathways, protein kinases/intracellular signaling, and the hedgehog pathway.
  • The review states that drugs that block abnormal functions of these pathways have been reported to remarkably improve survival in uLMS patients.
  • Chemotherapy resistance remains a major unmet need, motivating the search for novel drugs that effectively target these pathways.
  • The authors review emerging roles of epigenetics and the epitranscriptome, as well as serum markers, AI/machine learning approaches, shear wave elastography, current management options, and ongoing clinical trials.
Limitations: Narrative review rather than primary research — no original patient-level data presented.; Abstract does not state systematic review methods or a structured literature search, so selection bias in included literature is possible.; Broad scope synthesizes preclinical, early-phase, and clinical data together, which may mix evidence levels.; Survival ranges reported are wide and drawn from the literature rather than newly generated cohort data..

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed · Full text

Case reportMechanismReported positiveLimited evidenceTier 3 · early humann = 1

Elevated 68 Ga-FAPI Activity in Leiomyosarcoma

Clinical nuclear medicine · May 2024 · case report

leiomyosarcomaliver metastases

This is a case report of a 45-year-old woman with leiomyosarcoma who underwent 68 Ga-FAPI-04 PET/CT. The scan showed increased FAPI uptake in the abdominal primary tumor and in liver metastases. The authors state that 68 Ga-FAPI may have potential value in evaluating leiomyosarcoma.

Key findings
  • 68 Ga-FAPI-04 PET/CT detected increased FAPI uptake in abdominal leiomyosarcoma.
  • 68 Ga-FAPI-04 PET/CT detected increased FAPI uptake in liver metastases.
  • The authors suggest 68 Ga-FAPI may have potential value in the evaluation of leiomyosarcoma.
Limitations: Single-patient case report (n=1), limiting generalizability.; No quantitative imaging metrics (e.g., SUV) are reported in the abstract.; No comparison with other imaging modalities or controls presented in the abstract.; No longitudinal follow-up or clinical outcome data reported in the abstract..

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed

Case reportReported positiveLimited evidenceTier 3 · early humann = 1

Primary Gastric Leiomyosarcoma: A Rare Case

Cureus · Nov 2023 · case report

primary gastric leiomyosarcoma

This case report describes a single patient with a primary gastric leiomyosarcoma in the fundus/cardia. Pathology showed spindle cells with moderate to focally marked nuclear atypia, rare mitoses, positive immunoreactivity for smooth muscle actin and desmin, and negative CD117. The tumor was removed by laparoscopic partial gastrectomy; the patient fully recovered and had no recurrence or metastasis during seven years of follow-up. The authors also present a literature review on primary gastric leiomyosarcoma.

Key findings
  • Tumor located in the fundus/cardia region; tumoral spindle cells with diffusely moderate nuclear atypia, focally marked atypia, and rare mitotic figures.
  • Immunohistochemistry: positive for smooth muscle actin and desmin; negative for CD117 (c-kit).
  • Tumor was resected via laparoscopic partial gastrectomy with full recovery.
  • No recurrence or metastatic tumor detected during seven-year follow-up.
  • Authors conducted a literature review on primary gastric leiomyosarcoma.
Limitations: Single-patient case report — findings are not generalizable.; No control or comparator group and no systematic evaluation of treatments.; Abstract provides limited clinical details (e.g., demographics, perioperative care, adjuvant therapy not reported).; No molecular or genetic analyses beyond immunohistochemistry are reported in the abstract..

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed · Full text

Case reportInconclusiveLimited evidenceTier 3 · early humann = 1

Vulvar leiomyosarcoma: A case report

Gynecologic oncology reports · Jul 2023 · case report

vulvar leiomyosarcoma

This report describes a 35-year-old woman who presented with a right vulvar mass that was completely excised. Histopathology with immunohistochemistry diagnosed leiomyosarcoma, and the authors discuss contextual challenges that compromised her care and illustrate difficulties delivering safe cancer care in their setting.

Key findings
  • Vulvar leiomyosarcoma is a rare malignant smooth muscle tumor and the most common type of vulvar sarcoma that can mimic benign tumors, risking misdiagnosis.
  • A 35-year-old woman with a right vulvar mass underwent complete excision; histopathology with immunohistochemistry demonstrated leiomyosarcoma.
  • The authors highlight contextual challenges in their setting that ultimately compromised the patient's care and emphasize barriers to safe cancer care delivery.
Limitations: Single-patient case report limits generalizability.; No quantitative outcomes or follow-up data reported in the abstract.; No comparison group or systematic data collection.; Details of management beyond excision and subsequent clinical course are not provided..

AI summary of the abstract, human-reviewed · Jun 2026. Describes what this study reported, not medical advice. View on PubMed · Full text

Browse all studies mentioning Leiomyosarcoma

Where the evidence is

What has been studied, and how strong it is, by topic. A dashed cell means no studies were found for that combination — a gap, not evidence of no effect. Open a row to see its studies.

CompoundHuman evidenceMechanismSafetyTrial
Cisplatin11
Olaparib11
Trastuzumab Deruxtecan1
Trastuzumab-Deruxtecan (T-Dxd)1
Bevacizumab1
Dacarbazine1
Doxorubicin1
Gemcitabine
Ifosfamide1
Nivolumab †Rx
Pembrolizumab

Study mix

62 published studies by what they were done in. Lab and animal findings often do not carry over to people.

14 Human48 Review/other
Reported directionReported positive11Mixed results20Reported negative6Inconclusive25

Compounds with reported-positive results in Leiomyosarcoma

Where at least one study reported a positive result, shown with the full picture, not just the wins. Positive results are more likely to be published, and most of these are early lab or animal studies that may not translate to people. This reports what studies found, not what works.

Human evidence

Limitations: Retrospective, single-center design; Very small sample size (n=10); No control or comparator arm; Heterogeneous mix of gynecologic histologies; Heavily pre-treated population limits generalizability; Limited/absent reporting of safety or adverse event data in the abstract.
Cited positive studies (1)
Trastuzumab Deruxtecan1 positive1 human
Limitations: Retrospective, single-center design; Very small sample size (n=10); No control or comparator arm; Heterogeneous mix of gynecologic histologies; Heavily pre-treated population limits generalizability; Limited/absent reporting of safety or adverse event data in the abstract.
Cited positive studies (1)
Olaparib1 positive1 human
Limitations: Small number of HRD cases (5 of 58), with only three patients having mature clinical follow-up.; Non-randomized, observational access to PARPi in patients (no control arm reported).; PDX (preclinical) findings may not fully predict clinical efficacy.; Sequencing methods varied across samples (WGS, WES, panel), and some genomic HRD metrics (CHORD) were discordant.; No dosing, schedule, or detailed treatment-response durations provided in the abstract..
Cited positive studies (1)
Cisplatin1 positive1 human
Limitations: Small number of HRD cases (5 of 58), with only three patients having mature clinical follow-up.; Non-randomized, observational access to PARPi in patients (no control arm reported).; PDX (preclinical) findings may not fully predict clinical efficacy.; Sequencing methods varied across samples (WGS, WES, panel), and some genomic HRD metrics (CHORD) were discordant.; No dosing, schedule, or detailed treatment-response durations provided in the abstract..
Cited positive studies (1)

Evidence at a glance: compounds studied in Leiomyosarcoma

A deterministic grade of what published studies report for each: strength of evidence, the reported direction, and the largest credible effect, strongest-evidence first. This summarizes findings; it is not a claim that anything works.

CisplatinHuman · observationalReported positive1 human

Human observational evidence only — no trials.

Largest credible effect: Individuals with HRD uLMS in the screened cohort 9%, n=58 PMID 37143137 · effect sizes 2–13 across 4 studies

Most authoritative study: Targeting homologous recombination deficiency in uterine leiomyosarcoma

Based on a single study.
OlaparibHuman · observationalReported positive1 human

Human observational evidence only — no trials.

Largest credible effect: Individuals with HRD uLMS in the screened cohort 9%, n=58 PMID 37143137 · effect sizes 2–13 across 4 studies

Most authoritative study: Targeting homologous recombination deficiency in uterine leiomyosarcoma

Based on a single study.
Trastuzumab DeruxtecanHuman · observationalReported positive1 human

Human observational evidence only — no trials.

Largest credible effect: median PFS 5.4 mo [0.8–9.8], n=10 PMID 39639215 · effect sizes 1–5 across 3 studies

Most authoritative study: Real-world evidence of Trastuzumab Deruxtecan (T-DXd) Efficacy in HER2-expressing gynecological malignancies

Based on a single study.
Trastuzumab-Deruxtecan (T-Dxd)Human · observationalReported positive1 human

Human observational evidence only — no trials.

Largest credible effect: median PFS 5.4 mo [0.8–9.8], n=10 PMID 39639215 · effect sizes 1–5 across 3 studies

Most authoritative study: Real-world evidence of Trastuzumab Deruxtecan (T-DXd) Efficacy in HER2-expressing gynecological malignancies

Based on a single study.
BevacizumabInsufficient evidenceInconclusive

No primary experimental studies yet.

Largest credible effect: objective response (complete + partial) 35%, n=69 PMID 29759566 · response rates 11–35 across 4 studies

Most authoritative study: Role of bevacizumab in uterine leiomyosarcoma

No human studies yet · Based on a single study.
DacarbazineInsufficient evidenceMixed results

No primary experimental studies yet.

Most authoritative study: Management of advanced uterine leiomyosarcoma

No human studies yet · No numeric effect sizes reported · Based on a single study.
DoxorubicinInsufficient evidenceMixed results

No primary experimental studies yet.

Most authoritative study: Management of advanced uterine leiomyosarcoma

No human studies yet · No numeric effect sizes reported · Based on a single study.
GemcitabineInsufficient evidenceReported negative

No primary experimental studies yet.

Largest credible effect: chemotherapy_duration 6 mo, n=1 PMID 39286855 · effect sizes 6–8 across 2 studies

Most authoritative study: Primary ovarian leiomyosarcoma: a case report

No human studies yet · Based on a single study.
IfosfamideInsufficient evidenceMixed results

No primary experimental studies yet.

Most authoritative study: Management of advanced uterine leiomyosarcoma

No human studies yet · No numeric effect sizes reported · Based on a single study.
Nivolumab †RxInsufficient evidenceMixed results

No primary experimental studies yet.

Most authoritative study: Uterine leiomyosarcoma: A review of the literature and update on management options

No human studies yet · No numeric effect sizes reported · Based on a single study.
PembrolizumabInsufficient evidenceMixed results

No primary experimental studies yet.

Most authoritative study: Uterine leiomyosarcoma: A review of the literature and update on management options

No human studies yet · No numeric effect sizes reported · Based on a single study.

What the research shows for Leiomyosarcoma

A plain-language summary of the reviewed studies OncoForge tracks for Leiomyosarcoma. It reports what those studies described, not a claim that any compound or therapy helps or harms Leiomyosarcoma. Most of this evidence is early, and findings often conflict.

  • Studies report that most published literature on leiomyosarcoma consists of narrative reviews, case reports, and retrospective series rather than large prospective clinical trials.
  • Studies report that for localized uterine leiomyosarcoma surgery (complete hysterectomy) is described as the mainstay of management in early-stage disease in multiple reviews.
  • Studies report that for advanced or metastatic leiomyosarcoma, conventional cytotoxic chemotherapy (anthracycline- or gemcitabine-based regimens) is commonly used but associated with limited median progression-free and overall survival in the reviewed literature.
  • Studies report a genomic sequencing effort of 167 uterine leiomyosarcomas that identified recurrent alterations and led to a proposed immunohistochemical panel (p53, Rb, PTEN, ATRX with follow-up markers) for classifying tumors in test and validation cohorts.
  • Studies report that retrospective data on cutaneous leiomyosarcoma (83 patients) show dermal tumors tend to be smaller, with low risk of metastasis and high 5-year overall survival in that cohort, whereas subcutaneous lesions had different behavior.

Supportive & alternative options discussed

  • Exercise / prehabilitation: Also discussed as a supportive option to help maintain physical function, reduce fatigue, and improve quality of life for people with sarcomas in general.
  • Acupuncture: Also discussed as a supportive option for symptom management (for example pain or treatment-related side effects) in cancer care, though not specifically established for leiomyosarcoma in these studies.
  • Mind–body (MBSR / CBT): Also discussed as a supportive option to address psychological distress and improve wellbeing for patients with cancer, including sarcoma patients in broader literature.
  • Mistletoe (VAE): Also discussed in some regional/integrative oncology contexts as a complementary therapy for cancer symptom support, but no leiomyosarcoma-specific evidence is reported in these studies.

What we don’t know yet

  • Dozens of narrative reviews and small series exist, but the studies do not establish which systemic therapies (beyond conventional chemotherapy) improve survival in leiomyosarcoma in randomized trials.
  • Which genomic or immunohistochemical biomarkers predict response to specific therapies in leiomyosarcoma remains unclear despite sequencing studies and proposed IHC algorithms.
  • There is insufficient prospective data on the safety, optimal dosing, and clinical benefit of newer targeted agents or antibody–drug conjugates specifically in leiomyosarcoma.
  • How findings from uterine or cutaneous leiomyosarcoma apply across anatomic subtypes (retroperitoneal, vascular, uterine, cutaneous) is uncertain because many reports focus on specific locations.
  • Long-term outcomes, quality-of-life effects, and comparative effectiveness of different multimodal strategies (surgery, radiation, systemic therapy) have not been established in high-quality prospective studies.
Overall, the evidence base for leiomyosarcoma summarized in these studies is mainly narrative reviews, retrospective series, and small observational cohorts, so findings are preliminary and heterogeneous.

Clinical trials in Leiomyosarcoma

21 ongoing · 51 completed · tracked from ClinicalTrials.gov. Recruiting is not the same as proven, and completed is not the same as positive — read the results. Not a recommendation.

Completed
9 stopped (terminated / withdrawn / suspended)

Search all trials on ClinicalTrials.gov →

Getting care & support

Nonprofit / Gov

Practical, vetted help for Leiomyosarcoma — advocacy, paying for treatment, second opinions, and caregivers.

If you’re struggling emotionally, you don’t have to wait.

Advocacy & community

No dedicated organization for this specific cancer is curated yet — these general organizations can help in the meantime.

Financial help

  • PAN FoundationCopay assistance funds by diagnosis (funds open and close as money allows). · status changes often — check the fund’s site
  • HealthWell FoundationCopay and premium assistance funds by disease. · status changes often — check the fund’s site
  • CancerCare — financial assistanceLimited grants plus free financial counseling. · status changes often — check the fund’s site
  • Family ReachHelp with everyday living costs (rent, transport, food) during treatment. · status changes often — check the fund’s site
  • NeedyMedsSearchable directory of drug patient-assistance and discount programs. · status changes often — check the fund’s site
What you’ll typically need to apply
  • Your diagnosis and, if you have it, the specific drug/treatment name (from your care team).
  • Insurance details — your member ID card, or a note that you're uninsured (some funds require active insurance, some don't).
  • Proof of income and household size (recent pay stubs, a tax return, or a benefits letter) — most funds are income-based.
  • Your prescriber's contact information; some programs need the clinic to submit part of the application.
  • Apply early and re-check: funds open and close as money is available, so a closed fund may reopen.

General guidance — each program sets its own eligibility. Confirm requirements on the program’s site.

Help paying for the medicines on this page

Second opinions

Caregiver support

We list only non-profit and government resources — never product sellers — and take no affiliate fees. If a link is broken or a resource doesn't meet that bar, tell us.

Interactions & safety to check: Leiomyosarcoma

This is not a complete interaction check. It only covers the compounds we track and the signals reported in studies. A drug or supplement not listed here is not therefore safe. Bring your full medication and supplement list to your pharmacist and oncologist before changing anything.

Potential interactions: highest-stakes first

  • Nivolumab †Rx×immunosuppressantshigh-stakeshigh
    Avoid: Blunts efficacy (e.g., chronic steroids).
  • Nivolumab †Rx×Ipilimumabmoderate
    Synergize: Higher irAE but OS gains in melanoma.
  • Nivolumab †Rx×corticosteroidslow
    Use For IrAE: High-dose for toxicity; low-dose physiologic OK.

Safety considerations

Heading to an appointment? Get a printable one-page summary — studied compounds, open trials, interactions, and questions to ask.
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